मनोचिकित्सक: क्लिनिकल और चिकित्सीय जर्नल

खुला एक्सेस

हमारा समूह 1000 से अधिक वैज्ञानिक सोसायटी के सहयोग से हर साल संयुक्त राज्य अमेरिका, यूरोप और एशिया में 3000+ वैश्विक सम्मेलन श्रृंखला कार्यक्रम आयोजित करता है और 700+ ओपन एक्सेस जर्नल प्रकाशित करता है जिसमें 50000 से अधिक प्रतिष्ठित व्यक्तित्व, प्रतिष्ठित वैज्ञानिक संपादकीय बोर्ड के सदस्यों के रूप में शामिल होते हैं।

ओपन एक्सेस जर्नल्स को अधिक पाठक और उद्धरण मिल रहे हैं
700 जर्नल और 15,000,000 पाठक प्रत्येक जर्नल को 25,000+ पाठक मिल रहे हैं

अमूर्त

Untreated Neuroborreliosis presenting with somatic delusions and subjective memory impairment

Setare Eslami

Lyme disease is a tick-borne spirochete illness with multi involvement. Although neurological manifestations of Lyme disease are becoming increasingly well recognized, its psychiatric presentations are not well known. Physicians need to be aware of its neuropsychiatric symptoms that may present months to years later. Endemic areas to BorelliaBurgdorferi also have higher rates of schizophrenia. Prompt diagnosis and effect treatment is needed to avoid irreversible neuropsychiatric complications. We present a case of a 63 year old male with a psychiatric history of somatic delusions, pressured speech, disinhibition, subjective cognitive complaints with compensatory behavior, and pain on the anterior aspect of the upper torso. Patient believed symptoms stem from pesticide accumulation in fat cells after a single use of pesticides several years prior to initial somatic symptoms. Patient attempted to self-treat with over-the and supplements and sought the help of several internists and specialists. Patient discontinued follow up with several providers due to belief that physicians did not understand the association between his pesticide use and current symptoms. Upon psychiatric evaluation MMSE and MOCA were performed and found to be within normal limits. Subsequent Frontal Assessment Battery was found to be at threshold. Contact with current primary care provider to discuss concerns regarding possible neurologic etiology. MRI of the brain was preformed that demonstrated a frontal lobe hyperintensity. chronic pain of the anterior upper torso and cognitive complaints without change in MOCA performance.